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Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
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Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
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Reappraisal of metabolic dysfunction in neurodegeneration: Focus on mitochondrial function and calcium signaling
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LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
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Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
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Frontiers | Endoplasmic Reticulum-Mitochondria Contacts: A Potential Therapy Target for Cardiovascular Remodeling-Associated Diseases
Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
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LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
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Reappraisal of metabolic dysfunction in neurodegeneration: Focus on mitochondrial function and calcium signaling
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Processes | Free Full-Text | Competing Endogenous RNAs in Cervical Carcinogenesis: A New Layer of Complexity | HTML
